Article Figures & Data
Figures
- Figure 1 Flowchart Presenting the Selection Process of Patients From the Study Center Database
*High-risk Abs other than Hu included Ma2 (n = 9), Yo (n = 3), SOX1 (n = 2), Ri (n = 1), and CV2/CRMP5 (n = 1). In 2 patients, coexistent Hu-Ab and, respectively, SOX1 and CRMP5/CV2 were present. Ab = antibody; N-irAE = neurologic immune-related adverse event.
- Figure 2 Representative Neuroimaging Findings
(A) Patient 2: onset, a) mild cerebellar atrophy. Follow-up, b) hyperintense signal involving the dentate nuclei and periventricular brainstem regions; c) hyperintense signal involving the anterior medulla; d) moderate cerebellar atrophy. (B) Patient 4: onset, a) T2-hyperintense signal involving the bilateral medial temporal lobes; b) contrast enhancement involving the right medial temporal lobe. Follow-up, c) moderate atrophy of the medial temporal lobes; d) cauda equina root contrast enhancement. (C) Patients 5: onset, a) T2-hyperintense signal involving the left medial temporal lobe; b) diffuse pachymeningeal enhancement; c) T2-hyperintense signal involving both orbitofrontal lobes. Follow-up, d) regression of the orbitofrontal lobe T2-hyperintense signal.
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